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Misrepair of DNA double

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Some results are removed in response to a notice of local law requirement.For more information,please see here.Previous123456NextLarge XPF-dependent deletions following misrepair of a DNA

Brustel,J.,Kozik,Z.,Gromak,N.et al.Large XPF-dependent deletions following misrepair of a DNA double strand break are prevented by the RNA:DNA helicase Senataxin.Sci Rep 8,3850 (2018 Some results are removed in response to a notice of local law requirement.For more information,please see here.12345NextStochastic multicellular modeling of x-ray irradiation Dec 11,2019 Misrepair of DNA double#0183;The repair or misrepair of DNA double-strand breaks (DSBs) largely determines whether a cell will survive radiation insult or die.A new computational model of multicellular,track structure-based Some results are removed in response to a notice of local law requirement.For more information,please see here.

Scientists Say Glyphosate DNA Damage May Cause Cancer

Oct 10,2017 Misrepair of DNA double#0183;Chromosomal translocations are a hallmark of non-Hodgkin lymphoma (NHL) and can arise as a consequence of misrepair of DNA double-strand breaks. Deidre A.Hill et al.,Risk of non-Hodgkin lymphoma (NHL) in relation to germline variation in DNA repair and related genes, Blood (2006);108 3161-3167.Regulation of repair pathway choice at two-ended DNA Oct 01,2017 Misrepair of DNA double#0183;A DNA double-strand break (DSB) is considered to be a critical DNA lesion because its misrepair can cause severe mutations,such as deletions or chromosomal translocations.For the precise repair of DSBs,the repair pathway that is optimal for the particular circumstance needs to be selected.Radiation-Induced Chromosome Aberrations inRadiation-induced chromosome aberrations,particularly exchange-type aberrations,are thought to result from misrepair of DNA double-strand breaks.The relationship between individual pathways of break repair and aberration formation is not clear.By electrophoretic karyotyping of single-cell clones derived from irradiated cells,we have analyzed the induction of stable aberrations in haploid

Radiation-Induced Chromosome Aberrations in

Radiation-induced chromosome aberrations,particularly exchange-type aberrations,are thought to result from misrepair of DNA double-strand breaks.The relationship between individual pathways of break repair and aberration formation is not clear.RNF8 promotes high linear energy transfer carbon-ion Jul 01,2020 Misrepair of DNA double#0183;1.Introduction.DNA double-stranded breaks (DSBs) are considered to be toxic and mutagenic DNA lesions that lead to genomic instability,because failure of DSB repair causes chromosomal aberrations,such as deletions,translocations,and dicentric chromosomes,which may lead to the development of cancer [,,,].In contrast,DSBs are also induced by radiotherapy and mostMyeloid Leukemias Have Increased Activity of the Human myeloid leukemias are characterized by chromosomal abnormalities,including translocations,deletions,and allelic loss.These alterations are known to disrupt the function of genes that contribute to tumor initiation and progression.The mechanism underlying the appearance of these chromosomal alterations is poorly understood.Recent evidence suggests that altered nonhomologous end

Myeloid Leukemias Have Increased Activity of the

Human myeloid leukemias are characterized by chromosomal abnormalities,including translocations,deletions,and allelic loss.These alterations are known to disrupt the function of genes that contribute to tumor initiation and progression.The mechanism underlying the appearance of these chromosomal alterations is poorly understood.Recent evidence suggests that altered nonhomologous end Monte Carlo Damage Simulation (MCDS) SoftwareResults from the MCDS have been cited in at least 41 peer-reviewed studies and 2 book chapters.In a 2012 Red Journal paper by Frese et al.2012,the MCDS was used in combination with the repair-misrepair-fixation model to predict spatial variations in the relative biological effectiveness (RBE) of protons and carbon ions in representative spreadout Bragg peaks (SOBP).Misrepair of Particulate Chromium(VI)-Induced DNA Double Xie,Hong,Misrepair of Particulate Chromium(VI)-Induced DNA Double Strand Breaks Leads to Neoplastic Transformation (2007).Electronic Theses and Dissertations .769.

Misrepair of DNA double-strand breaks after exposure to

May 30,2013 Misrepair of DNA double#0183;Misrepair of DNA double-strand breaks after exposure to heavy-ion beams causes a peak in the LET-RBE relationship with respect to cell killing in DT40 cells.Mizuho Aoki-Nakano School of Allied Health Sciences,Kitasato University,1-15-1 Kitasato,Minami-ku,Misrepair of DNA double-strand breaks after exposure to May 30,2013 Misrepair of DNA double#0183;Double-strand breaks (DSBs) are considered to be the most critical among the various types of DNA damage that can be generated by ionizing radiation.In eukaryotes,two major DSB repair pathways,which differ in their requirements for DNA homology,have been identified homologous recombination repair (HRR) and non-homologous end-joining (NHEJ).Misrepair Article about misrepair by The Free DictionaryMisrepair of radiation-induced DNA double-strand breaks and its relevance for tumorigenesis and cancer treatment (review).Health effects of ionizing radiation from diagnostic CT imaging Consideration of alternative imaging strategies.Misrepair can cause mutation,and mutants may be the start of a tumor.

Misrepair Article about misrepair by The Free Dictionary

Misrepair of radiation-induced DNA double-strand breaks and its relevance for tumorigenesis and cancer treatment (review).Health effects of ionizing radiation from diagnostic CT imaging Consideration of alternative imaging strategies.Misrepair can cause mutation,and mutants may be the start of a tumor.Large XPF-dependent deletions following misrepair of a DNA Brustel,J.,Kozik,Z.,Gromak,N.et al.Large XPF-dependent deletions following misrepair of a DNA double strand break are prevented by the RNA:DNA helicase Senataxin.Sci Rep 8,3850 (2018 Interpretation of the shape of survival curves in terms of Abstract.Evidence is presented that in yeast cells one DNA double-strand break (dsb) may be considered as one potentially lethal lesion (PLL).Using a temperature conditional radiosensitive diploid yeast mutant (rad 54-3) it is demonstrated that the shoulder of survival curves,for cells plated immediately,is due to repair of dsb (PLL) within a restricted time period.

Interpretation of the shape of survival curves in terms of

Abstract.Evidence is presented that in yeast cells one DNA double-strand break (dsb) may be considered as one potentially lethal lesion (PLL).Using a temperature conditional radiosensitive diploid yeast mutant (rad 54-3) it is demonstrated that the shoulder of survival curves,for cells plated immediately,is due to repair of dsb (PLL) within a restricted time period.Frontiers Misrepair in Context TGF Regulation of DNA Aug 27,2019 Misrepair of DNA double#0183;Repair of DNA damage protects genomic integrity,which is key to tissue functional integrity.In cancer,the type and fidelity of DNA damage response is the fundamental basis for clinical response to cytotoxic therapy.Here we consider the contribution of transforming growth factor-beta (TGF),a ubiquitous,pleotropic cytokine that is abundant in the tumor microenvironment,toDNA double-strand break repair in parental chromatin of INTRODUCTION.Both cell functioning and the environment pose threats to the integrity of the genetic information of the cell.A nucleus can be confronted with a variety of potentially genotoxic damages to which an intricate set of DNA repair mechanisms has evolved ( 1).A DNA double-strand break (DSB) is the most toxic lesion and is mainly repaired by either non-homologous end joining (NHEJ) or

Combined Use of Monte Carlo DNA Damage Simulations

Carlson,D.J.,Stewart,R.D.,Semenenko,V.A.and Sandison,G.A.Combined Use of Monte Carlo DNA Damage Simulations and Deterministic Repair Models to Examine Putative Mechanisms of Cell Killing.Radiat.Res.169,447459 (2008).A kinetic repair-misrepair-fixation (RMF) model is developed to better link double-strand break (DSB) induction to reproductive cell death.Formulas linking Cited by 3Publish Year 2018Author Chen-Chun Pai,Elizabeth Blaikley,Timothy C.HumphreyMisrepair of DNA double-strand breaks after exposure to Misrepair of DNA double-strand breaks after exposure to heavy-ion beams causes a peak in the LETRBE relationship with respect to cell killing in DT40 cells Mizuho Aoki-Nakano 1 and Yoshiya Furusawa 2,* 1 School of Allied Health Sciences,Kitasato University,1-15-1 Kitasato,Minami-ku,Sagamihara-shi,Kanagawa 252-0373,JapanCited by 2Publish Year 2019Author Jake C.Forster,Michael J.J.Douglass,Michael J.J.Douglass,Wendy M.Phillips,Wendy M.PhillipFrontiers Misrepair in Context TGF Regulation of DNA Aug 27,2019 Misrepair of DNA double#0183;DNA repair is executed by multiple pathways that must be coordinated to deal with different types of DNA damage,including oxidative damage,single strand breaks (SSB),and double strand breaks (DSB).Complex intracellular mechanisms have developed to ensure an appropriate DNA damage response (DDR).

Cited by 10Publish Year 2013Author Mizuho Aoki-Nakano,Yoshiya FurusawaMisrepair of radiation-induced DNA double-strand breaks

The faithful repair of DNA double-strand breaks (DSBs) is probably one of the most critical tasks for a cell in order to maintain its genomic integrity since these lesions may lead to chromosome breaks or rearrangements,mutations,cell death or cancer.Cited by 10Publish Year 2013Author Mizuho Aoki-Nakano,Yoshiya FurusawaDNA Double-Strand Break Repair AssayDNA double-strand breaks (DSBs),arising during normal DNA metabolism or following exposure to mutagenic agents such as ionizing radiation can lead to chromosomal rearrangements and genome instability,and are potentially lethal if unrepaired.Cited by 103Publish Year 2002Author Kai Rothkamm,Markus LobrichStochastic multicellular modeling of x-ray irradiation Dec 11,2019 Misrepair of DNA double#0183;The repair or misrepair of DNA double-strand breaks (DSBs) largely determines whether a cell will survive radiation insult or die.

Canonical DNA non-homologous end-joining; capacity

Nov 18,2019 Misrepair of DNA double#0183;Early studies on DNA double strand break (DSB) repair in prokaryotes and lower eukaryotes demonstrated that homologous recombination (HR) is the major DSB repair pathway and HR-deficient mutants display high radiation (IR)-sensitivity.1 Surprisingly,IR-sensitive and DSB repair defective mammalian cell lines proved to be proficient for HR but COREInterpretation of the shape of survival curves in terms of induction and repair/misrepair of DNA double-strand breaks.By D.Frankenberg,M.Frankenberg-Schwager and R.Harbich.Abstract.Evidence is presented that in yeast cells one DNA double-strand break (dsb) may be considered as one potentially lethal lesion (PLL).Using a temperature Bleomycin Side Effects Common,Severe,Long Term - DrugsDec 30,2019 Misrepair of DNA double#0183;The most frequent mutations produced in mammalian cells are large multilocus deletions,which probably arise by misrepair of (DNA) double strand breaks.Uncommon (0.1% to 1%) Tumor pain .Hematologic.Common (1% to 10%) Hemorrhage.Uncommon (0.1% to 1%) Leucopenia.Frequency not reported Pancytopenia,thrombocytopenia,anemia,neutropenia

Analysis of radiation-induced DNA double-strand breaks

Jun 11,2003 Misrepair of DNA double#0183;It has been suggested that the technique for measuring repair fidelity of radiation-induced DNA double-strand breaks (DSBs) using Southern blotting and hybridization to defined regions of the genome could be compromised by broken or poorly-digested DNA.Since misrepair of DNA DSBs is an important aspect of radiation-induced chromosome aberrations,mutations,and cell killing,weA role for p53 in DNA end rejoining by human cell extracts The tumor suppressor p53 is a major regulator in the response of human cells to DNA damage.In this study we assessed the role of p53 in the repair of DNA double-strand breaks in plasmid DNA using cell extracts from three human lymphoblastoid cell lines derived from the same donor.(PDF) Misrepair of DNA double-strand breaks afterMisrepair of DNA double-strand breaks after exposure to heavy-ion beams causes a peak in the LETRBE relationship with respect to cell killing in DT40 cells.pdf Available via license CC BY 3.0

(PDF) Misrepair of DNA double-strand breaks after

Misrepair of DNA double-strand breaks after exposure to heavy-ion beams causes a peak in the LETRBE relationship with respect to cell killing in DT40 cells.pdf Available via license CC BY 3.0 (PDF) Large XPF-dependent deletions following misrepair of Large XPF-dependent deletions following misrepair of a DNA double strand break are prevented by the RNA:DNA helicase Senataxin Article (PDF Available) in Scientific Reports 8(1) December 2018 (PDF) Large XPF-dependent deletions following misrepair of Large XPF-dependent deletions following misrepair of a DNA double strand break are prevented by the RNA:DNA helicase Senataxin Article (PDF Available) in Scientific Reports 8(1) December 2018

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